I just had to copy this very interesting - and hard to find anywhere else- information about binge eating from youreatopia.com. Link to the whole page is found at the bottom.
Bingeing in the true clinical sense only occurs for the tiny group suffering what appear to be rare circadian rhythm and endocrine disorders that result in binge eating disorder or night eating syndrome.
Critically, those with BED are not able to apply any restrictive responses to their overeating. The condition appears in pre-pubescent childhood and usually involves being classified as ‘obese’ by age 11.
I’m going to repeat that:
Binge eating as a clinical disorder involves an inability to apply any restriction.
That means, if you are on the restriction eating disorder spectrum, you are unable to develop BED. Your bingeing experience is an expression of required energy needs in reaction to restrictive eating behaviors. It is why I call this behavior reactive eating and not bingeing.
The answer is “no”. However, it is important to note that the process of recovery from an energy deficient state most certainly involves a period of extreme hunger and eating. It is a transient condition that disappears once energy balance is restored.
Here are the likely reasons why those who reach a complete remission from a restrictive eating disorder do not develop binge eating disorder:
No one keeps gaining and gaining. We each have an optimal weight set point [RE Keesey et al., 1997; RE Keesey, 1988]. On average 70% of adult females reside between BMI 21-27 [Statistics Canada, 1978] but our heights and weights exist on a bell curve and you are only going to be healthy at your particular optimal weight set point.
Non-ED men and women ‘overeat’ regularly and it is not bingeing in any clinical sense nor does it impact optimal weight stability.
Between the ages of 10-16 it is common for the body to store extra energy in anticipation of physical growth requirements. It is difficult for anyone in our obese-fearing and weight-obsessed culture to not react to these phases of extra weight with immediate restriction. Sadly, for those with the restrictive eating disorder genotype, it is usually this very circumstance that catapults them into years of cycling through restrictions and quasi-recoveries.
Between the ages of 16-25, the body will occasionally store extra energy, but usually it is using the extra energy coming in (through natural overeating sessions) as it happens. However, if a restrictive eating disorder patient hijacked his or her normal development as a child with self-administered starvation and/or excessive exercise, then the recovery process may mimic the energy storage/growth spurt that was supposed to happen but was stalled by the onset of the eating disorder. Give it time and it works itself out.
The answer to getting out of this quasi-recovered state and reaching full recovery is to eat to the recovery guideline amounts (and more) every single day. Responding to any extreme hunger is as critical now as it is all throughout the recovery process.
Despite restrictive eating disorder-based fears that there is no way to eat this much and not become obese when you are already ‘weight restored’, no clinical evidence supports those fears.
Bingeing in the true clinical sense only occurs for the tiny group suffering what appear to be rare circadian rhythm and endocrine disorders that result in binge eating disorder or night eating syndrome.
Critically, those with BED are not able to apply any restrictive responses to their overeating. The condition appears in pre-pubescent childhood and usually involves being classified as ‘obese’ by age 11.
I’m going to repeat that:
Binge eating as a clinical disorder involves an inability to apply any restriction.
That means, if you are on the restriction eating disorder spectrum, you are unable to develop BED. Your bingeing experience is an expression of required energy needs in reaction to restrictive eating behaviors. It is why I call this behavior reactive eating and not bingeing.
Will I Become A Binge-Eater?
Does someone who recovers from the restrictive eating disorder, no matter the restrictive facets they experience, develop a binge eating disorder?The answer is “no”. However, it is important to note that the process of recovery from an energy deficient state most certainly involves a period of extreme hunger and eating. It is a transient condition that disappears once energy balance is restored.
Here are the likely reasons why those who reach a complete remission from a restrictive eating disorder do not develop binge eating disorder:
- The candidate genes associated with inflammatory obesity, cholesterol, insulin and glucose levels are unrelated to the genes identified thus far for restrictive eating disorders [R Stöger, 2012; AW Drong et al., 2012; CJ Nolan et al., 2011; A. Hinney et al., 1999 and 2000].
- Patients fully recovered from the restrictive eating disorder rarely reach final restored weights above BMI 25. 2% of the recovered population does go above BMI 25, however ALL return to weights at or below BMI 25 after one full year beyond recovery (with no relapse of restriction involved) [CM Bulik et al., 2006].
- Leptin resistance is not a factor in either the activation or resolution of a restrictive eating disorder. Yet, leptin resistance is often present in patients with inflammatory obesity. [P. Dandona et al., 2004; JF Caro et al., 1996; S Herpertz et al., 1998 and 2000]
No one keeps gaining and gaining. We each have an optimal weight set point [RE Keesey et al., 1997; RE Keesey, 1988]. On average 70% of adult females reside between BMI 21-27 [Statistics Canada, 1978] but our heights and weights exist on a bell curve and you are only going to be healthy at your particular optimal weight set point.
Non-ED men and women ‘overeat’ regularly and it is not bingeing in any clinical sense nor does it impact optimal weight stability.
Between the ages of 10-16 it is common for the body to store extra energy in anticipation of physical growth requirements. It is difficult for anyone in our obese-fearing and weight-obsessed culture to not react to these phases of extra weight with immediate restriction. Sadly, for those with the restrictive eating disorder genotype, it is usually this very circumstance that catapults them into years of cycling through restrictions and quasi-recoveries.
Between the ages of 16-25, the body will occasionally store extra energy, but usually it is using the extra energy coming in (through natural overeating sessions) as it happens. However, if a restrictive eating disorder patient hijacked his or her normal development as a child with self-administered starvation and/or excessive exercise, then the recovery process may mimic the energy storage/growth spurt that was supposed to happen but was stalled by the onset of the eating disorder. Give it time and it works itself out.
The answer to getting out of this quasi-recovered state and reaching full recovery is to eat to the recovery guideline amounts (and more) every single day. Responding to any extreme hunger is as critical now as it is all throughout the recovery process.
Despite restrictive eating disorder-based fears that there is no way to eat this much and not become obese when you are already ‘weight restored’, no clinical evidence supports those fears.
Your
metabolism will ensure that your body adjusts to its optimal weight set
point and the excess energy is necessarily needed to complete the
lingering repair and to finally push the metabolism back to its optimal
functioning rate. And no, your metabolism is not broken and your brain
responds accurately to leptin levels.
As for whether the temporary overshoot is necessary, there is some evidence that it may indeed have value in ensuring the return of an optimal fat-mass to fat-free-mass ratio. In fact, Abdul Dulloo and colleagues re-examined the Minnesota trial data and discovered that the depletion of fat-free mass and fat mass (occurring during starvation) separately trigger hyperphagia (excessive eating) in post-starvation subjects and that the hyperphagia will persist until both fat and fat-free mass are restored [A Dulloo et al., 1997].
We also know from numerous other studies that anorexics often maintain a higher proportion of fat mass post-re-feeding [CI Orphanidou et al., 1997; M Probst et al., 2001; C Mantzoros et al., 1997] and this is likely due to the prevailing attitudes that hyperphagia must be avoided during recovery at all costs as it is considered a marker of “bingeing”. Instead, what these post-recovery data may show is that the prohibition of hyperphagia in recovery from restrictive eating disorders serves to halt the body’s ability to return to an optimal fat mass to fat-free mass ratio.
As for whether the temporary overshoot is necessary, there is some evidence that it may indeed have value in ensuring the return of an optimal fat-mass to fat-free-mass ratio. In fact, Abdul Dulloo and colleagues re-examined the Minnesota trial data and discovered that the depletion of fat-free mass and fat mass (occurring during starvation) separately trigger hyperphagia (excessive eating) in post-starvation subjects and that the hyperphagia will persist until both fat and fat-free mass are restored [A Dulloo et al., 1997].
We also know from numerous other studies that anorexics often maintain a higher proportion of fat mass post-re-feeding [CI Orphanidou et al., 1997; M Probst et al., 2001; C Mantzoros et al., 1997] and this is likely due to the prevailing attitudes that hyperphagia must be avoided during recovery at all costs as it is considered a marker of “bingeing”. Instead, what these post-recovery data may show is that the prohibition of hyperphagia in recovery from restrictive eating disorders serves to halt the body’s ability to return to an optimal fat mass to fat-free mass ratio.
http://www.youreatopia.com/blog/2012/10/31/bingeing-is-not-bingeing.html
also take a look at http://www.youreatopia.com/blog/2011/11/3/binge-eating-disorder-and-night-eating-syndrome.html
also take a look at http://www.youreatopia.com/blog/2011/11/3/binge-eating-disorder-and-night-eating-syndrome.html
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